jmp version 3.0.2 software program (SAS institute)
90
Structured Review
SAS institute
jmp version 3.0.2 software program
Jmp Version 3.0.2 Software Program, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/jmp+version+3%2E0%2E2+software+program/jmp+software+package/pm11408828-81-13-18
Average 90 stars, based on 1 article reviews
Jmp Version 3.0.2 Software Program, supplied by SAS institute, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/jmp+version+3%2E0%2E2+software+program/jmp+software+package/pm11408828-81-13-18
Average 90 stars, based on 1 article reviews
jmp version 3.0.2 software program - by Bioz Stars,
2026-09
90/100 stars
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Comparison:Article Title: Tas-301, a new synthetic inhibitor of neointimal thickening after balloon injury, inhibits calcium-dependent signal transduction and cytoskeletal reorganization. Article Snippet: We previously demonstrated that a recently synthesized drug, TAS-301 [3-bis(4-methoxyphenyl)methylene-2-indolinone], inhibited neointimal thickening after singleballoon injury to the rat common carotid artery by inhibiting both the migration and proliferation processes of vascular smooth muscle cells (VSMCs).. The purpose of this current study was to elucidate the possible mechanism of action for its inhibition of the migration process of VSMCs.. We also determined the efficacy of TAS-301 on second neointimal formation 14 days after a doubleballoon injury to the rat common carotid artery. Software:Article Title: Tas-301, a new synthetic inhibitor of neointimal thickening after balloon injury, inhibits calcium-dependent signal transduction and cytoskeletal reorganization. Article Snippet: We previously demonstrated that a recently synthesized drug, TAS-301 [3-bis(4-methoxyphenyl)methylene-2-indolinone], inhibited neointimal thickening after singleballoon injury to the rat common carotid artery by inhibiting both the migration and proliferation processes of vascular smooth muscle cells (VSMCs).. The purpose of this current study was to elucidate the possible mechanism of action for its inhibition of the migration process of VSMCs.. We also determined the efficacy of TAS-301 on second neointimal formation 14 days after a doubleballoon injury to the rat common carotid artery. |